Over 55 million people worldwide have dementia.
, making it unlikely that semaglutide directly activates memory networks. So how might the drug treat the disease?
The brains of Alzheimer's disease patients accumulate sticky plaques of amyloid-beta as well as clumps of tau proteins inside brain cells. These are thought to disrupt cognition.is underway here at the University of Oxford that will specifically examine tau levels in people who have high levels of amyloid-beta but have not developed dementia. It is hoped that semaglutide will reduce cortical tau levels, leading to reduced rates of cognitive decline.target amyloid-beta plaques.
These types of drugs probably also target amyloid-beta around blood vessels, leading to potentially life-threatening such as bleeds in the brain. Alternative strategies are therefore desirable, and this is where semaglutide may shine. frameborder="0″ allow="accelerometer; autoplay; clipboard-write; encrypted-media; gyroscope; picture-in-picture; web-share" allowfullscreen>
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