Women are diagnosed with this common condition three times more often than men but in most cases treatment is trial and error. Why?
developed from the research of people like Griffiths. These drugs work by suppressing a peptide that soars in migraine patients during an attack.
"Where I'm at now is thinking about going back to work, knocking out my PhD, having a life again," she says. "I'm making friends. Life is good."In the past 20 years at least 50 genes have been found that to relate to migraine. For Crosby, while her genes drive the condition, her triggers include dust and preservative 202 which she calls "my kryptonite": "If I have even the slightest amount, I'm in all kinds of trouble".
"I suspect you'd like a scan of your brain?" the doctor deadpanned when I went for a check-up. The all-clear meant one thing was most likely: the migraines were back. Wijaratne now has around 17 different medications to choose from when treating patients — ranging from triptans to CGRPs, but he says coming up with an effective dose and combination remains trial and error.So where are the research funds?
This is a comparison Crosby can relate to having also experienced what she describes as "a minor skirmish with breast cancer". A mutation on the gene methylenetetrahydrofolate reductase can make carriers susceptible to migraine with aura, Griffiths says, and this gene reacts strongly to folate levels in the diet.
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